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About

Switching between corticosteroids is a routine but high-stakes task in clinical practice. The primary challenge lies in the variations of biological half-life, mineralocorticoid activity (salt-retention), and anti-inflammatory potency. For instance, Dexamethasone lacks mineralocorticoid activity but has a long biological half-life, making it biologically distinct from Hydrocortisone, which closely mimics endogenous cortisol.

This tool ensures therapeutic continuity during drug transitions. It uses the standard equivalency ratios accepted in pharmacology. Accuracy is paramount here. An incorrect conversion can lead to adrenal crisis (under-dosing) or Cushingoid side effects (over-dosing). This calculator normalizes dosages against a Hydrocortisone baseline to derive the precise equivalent volume for the target agent.

steroids prednisone dosage converter anti-inflammatory pharmacology

Formulas

Calculation relies on linear proportionality relative to the standardized equivalent base doses (Base). The formula to find the TargetDose is:

Dosetarget = Dosecurrent × BasetargetBasecurrent

Where Base represents the amount of drug equivalent to 20 mg of Hydrocortisone. For example, if converting from Prednisone (Base = 5) to Dexamethasone (Base = 0.75):

Dosedex = Dosepred × 0.15

Reference Data

DrugEquiv. Dose (mg)Anti-inflam. PotencyMineralo. PotencyHalf-life (hrs)
Hydrocortisone20118 - 12
Prednisone540.812 - 36
Prednisolone540.812 - 36
Methylprednisolone450.512 - 36
Triamcinolone45012 - 36
Dexamethasone0.7525036 - 72
Betamethasone0.625 - 30036 - 72

Frequently Asked Questions

No. The calculator focuses on anti-inflammatory equivalence. If switching from a drug with high mineralocorticoid activity (like Hydrocortisone or Fludrocortisone) to one with zero activity (like Dexamethasone), the patient may require separate mineralocorticoid replacement (e.g., Fludrocortisone) to maintain blood pressure and electrolyte balance.
No. This tool is strictly for systemic corticosteroids (oral or intravenous). Topical steroid potency relies on vasoconstrictor assays and formulation (ointment vs. cream), which uses a completely different classification system (Classes I-VII).
Yes, these are the standard pharmacological ratios for HPA axis suppression and anti-inflammatory effects. However, individual patient metabolism (e.g., liver function) can alter bioavailability, so clinical monitoring is always required after a switch.